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Ly transnational world, understanding the significance of immunological biomarkers such as CRP, in both clinical and non-clinical settings, will require integrating knowledge of how different ecological contexts influence the manifestation of non-acute immune function. While working with a subsistence agricultural population of pubertal girls has important benefits for understanding diversity in i
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He expression of distinct and likely overlapping gene sets. It seems plausible that the transcriptome of affected individuals reflects contributions from both the genetic background (genotype) and from environmentally mediated changes to the developmental transcription program. In other words, even though ASD is considered a complex genetic disorder, the complexity lies not only in the genetics, b
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N genes that overlap with chromatin remodeling proteins, transcriptional regulators and synapse-associated proteins [12] . Interestingly, these genes are also targets of environmentally induced changes in gene expression. ASD genetics: background During the last decade, it has become increasingly clear that in addition to SNPs, copy number variants (CNVs) account for a significant percentage of AS
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E MRC/DFID Concordat agreement. Funding to pay the Open Access publication charge for this article was provided by Harvard University. Conflict of interest: None declared.PerspectiveFor reprint orders, please contact: reprints@futuremedicine.comMerging data from genetic and epigenetic approaches to better understand autistic spectrum disorderAutism spectrum disorder (ASD) is a complex neurod
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E MRC/DFID Concordat agreement. Funding to pay the Open Access publication charge for this article was provided by Harvard University. Conflict of interest: None declared.PerspectiveFor reprint orders, please contact: reprints@futuremedicine.comMerging data from genetic and epigenetic approaches to better understand autistic spectrum disorderAutism spectrum disorder (ASD) is a complex neurod
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E MRC/DFID Concordat agreement. Funding to pay the Open Access publication charge for this article was provided by Harvard University. Conflict of interest: None declared.PerspectiveFor reprint orders, please contact: reprints@futuremedicine.comMerging data from genetic and epigenetic approaches to better understand autistic spectrum disorderAutism spectrum disorder (ASD) is a complex neurod
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E MRC/DFID Concordat agreement. Funding to pay the Open Access publication charge for this article was provided by Harvard University. Conflict of interest: None declared.PerspectiveFor reprint orders, please contact: reprints@futuremedicine.comMerging data from genetic and epigenetic approaches to better understand autistic spectrum disorderAutism spectrum disorder (ASD) is a complex neurod
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E MRC/DFID Concordat agreement. Funding to pay the Open Access publication charge for this article was provided by Harvard University. Conflict of interest: None declared.PerspectiveFor reprint orders, please contact: reprints@futuremedicine.comMerging data from genetic and epigenetic approaches to better understand autistic spectrum disorderAutism spectrum disorder (ASD) is a complex neurod