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Sociated with SLE susceptibility have been shown to contribute to high IFN levels in human SLE patients [21?23]. These data support the idea that gain-of-function polymorphisms in the IFN pathway are a common pathogenic mechanism in SLE. Additionally, number novel genetic loci have been identified that have an effect on IFN- levels in SLE patients, supporting the genetic nature of the IFN dysregul
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