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S is supported by the wide range of phenotypes described for hypomorphic and null mutants of CUL1. While complete loss of the cullin is embryo-lethal and causes abortion of embryogenesis at the early globular stage, hypomorphic mutants show pleiotropic defects related to lateral organ initiation, flower development, photomorphogenesis, and auxin and jasmonate signaling, as well as the circadian cl

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